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The effects of air hypobaria on inducible smoking-related responses have not been previously investigated. The purpose of this study was to explore the cardiovascular responses to cigarette smoke inhalation (endogenous nicotine and nicotine-related compounds) in man during normoxia and air hypobaria in order to examine whether the magnitude of these responses are altered by the presence of a non-NO2-related inhalant. In a double blind study, 10 non-smokers were exposed to cigarette smoke (total particulate matter concentration 0.8 mg m(-3)) from two conventional cigarettes under normoxic and hypobaric (ATA 760 torr) conditions. Inhalation was performed through a demand valve regulated at an inspired minute volume of 10 l.min(-1) for the duration of 10 min. Mean arterial pressure, heart rate, cardiac output and heart rate variation, plasma nicotine and cotinine were measured. Hypobaric air exposure had no significant effect on any of the measured responses to cigarette smoke inhalation. In contrast, hypobaria induced a significant increase in the resting heart rate and heart rate variation for both nicotine and saline control, changes which were mainly due to increases in the preexposure value. Mean arterial pressure was unaltered by air hypobaria. The effect of hypobaric air exposure on the sympathomimetic responses to cigarette smoke inhalation was not mediated by nicotine.This is the first book on the use of thiol chemistry in organic synthesis. It describes the preparation of novel compounds with varying thiol functionalities, as well as their biological properties. The reactions covered are metalations, electrophilic displacements and homologations. In addition, all the reactions which utilize dithioesters or sulfides are reviewed. The chapters are written to be accessible to organic chemists, with examples and detailed descriptions of the reactions that are used in laboratory practice. A fully revised and updated second edition is now available./*
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